@article{oai:ir.kagoshima-u.ac.jp:00005104,
 author = {Kitano, Motoo and 北野, 元生 and Urago, Atsushi and 浦郷, 篤史 and Hayashi, Ko and Kawashima, Mahito and Funakoshi, Keisuke and Yamada, Kunio and Tokufuji, Shin-ichiro},
 issue = {2},
 journal = {南太平洋研究, South Pacific Study},
 month = {Mar},
 note = {Histopathological analysis was done on brains of four autopsy cases of acute decompression
sickness (DCS). The findings of the brains in four cases of acute DCS was summarized as follows:
1) marked engorgement of the meningeal veins with marked stasis of blood with or without
presence of intravascular air bubbles, 2) marked edema of the cerebral parenchyma with
perivascular hemorrhage, 3) pallor spots around the venules and blood capillaries in the brain,
especially in the deeper cortex and subcortical white matter, and 4) relatively-sharply defined foci
of edematous necrosis in the deep layer of the white matter of the cerebrum, especially in the
periventricular layer.
As for the causes of the cerebral lesions in DCS, bubble embolization seemed to be the most
important factor. Alteration of permeability of the blood vessels caused by trapping of very
small bubble emboli should contribute significantly to the histogenesis of pallor spots.
Pathological-anatomical analysis ascribed that the pathogenesis of focal edematous necrosis to
marked disturbance of venous circulation due to bubble embloization with a superimposing severe
shock. For the latter lesion of the brain, the etiology seemed identical with that of the spinal cord
lision in DCS with apparent distruptions in the circulation of veins in and around the spinal cord.},
 pages = {275--285},
 title = {A Pathological Study on Cerebral Lesions in Diver's Decompression Sickness (DCS)},
 volume = {10},
 year = {1990},
 yomi = {キタノ, モトオ and ウラゴウ, アツシ}
}